2 edition of Endothelium-dependent hyperpolarizations found in the catalog.
International Symposium on Endothelium-Derived Hyperpolarizing Factor (2nd 1998 Cernay-la-Ville, France)
|Statement||edited by Paul M. Vanhoutte.|
|Contributions||Vanhoutte, Paul M.|
|LC Classifications||QP88.45 .E54x 1999|
|The Physical Object|
|Pagination||xxv, 436 p. :|
|Number of Pages||436|
Cardiac Surgery in the Adult, Fourth Edition. An update of the most recent surgical perspectives and techniques in ischemic and valvular heart disease, diseases of . Role of gap junctions in endothelium-dependent hyperpolarizations 7. Heterogeneity of EDHF-type relaxations of rabbit and rat arteries analysed with peptides homologous to the extracelluar loops of connex 40 and 43 8. Myoendothelial and circumferential spread of endothelium-dependent hyperpolarization in coronary arteries 9.
Emergency War Surgery Book - Written for the US Army. Information Filled pages% Off! Special SALE PRICE!Emergency War Surgery - Completely revised and updated edition. Contains 33 chapters of life saving information that could save a life in a no doctor situation. Electrophysiological recordings of membrane potentials with the microelectrode technique in mesenteric arteries demonstrated that endothelium-dependent hyperpolarizations to ACh (10 −5 M) in male mice in the presence of indomethacin (10 −5 M) and l-NNA (10 −4 M) were also progressively attenuated in accordance with the number of disrupted Cited by:
muscle and endothelium-dependent hyperpolarizations are not affected by inhibitors of NO synthasess or by oxyhaemoglobir?. Besides NO, the production of cGMP can also be increased by each of carbon monoxide, hydrogen perox- ide and hydroxyl radicals22, and these mediators could. Introduction. The endothelium maintains vascular homeostasis through the release of active gh Nitric Oxide (NO) is recognized as the primary factor at level of arteries, increased evidence for the role of another endothelium-derived vasodilator known as endothelium-derived hyperpolarizing factor (EDHF) has accumulated in the last years.
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Chapter 1 Endothelium-Dependent Hyperpolarizations: The Classical “EDHF” Pathway A lot of confusion has arisen in the field of endothelium-dependent hyperpolarizations due to the use of the acronym “EDHF” which implies that a single diffusible substance underlies this novel mechanism of relaxation of the vascular by: 1.
The first two meetings established a role for EDHF and other chemical factors involved in endothelium-dependent hyperpolarizations. These proceedings focus upon the role of gap junctions in cardiovascular responses and investigate the chemical identity of : Hardcover.
This monograph reports on the work of many researchers. It summarizes the significant recent discoveries concerning endothelium-dependent hyperpolarizations, which are likely to play a much more important role in cardiovascular physiology and pathology than was originally foreseen Extensively illustrated with original diagrams and schematics Cited by: This monograph reports on the work of many researchers.
It summarizes the significant recent discoveries concerning endothelium-dependent hyperpolarizations, which are likely to play a much more important role in cardiovascular physiology and pathology than was originally foreseen.
Extensively illustrated with original diagrams and schematics Cited by: Endothelium-dependent Endothelium-dependent hyperpolarizations book cannot be explained by electrical coupling between the endothelial and the smooth cells in muscular arteries; epoxyeicosatrienoic acids as endothelium-derived hyperpolarization factors in coronary arteries - potassium channel activation through endogenous ADP - ribosylation of Gs; phospholipid-derived epoxyeicosatrienoic acids mediate the relaxation's attributed to endothelium-derived hyperpolarizing factor; hyperpolarizing.
It reviews why endothelium-derived hyperpolarizing factor (EDHF) is not likely to be either NO or prostacyclin. It sets the stage for the other contributions of this special issue devoted to EDHF that will discuss in depth the current knowledge concerning endothelium-dependent by: These endothelium-dependent contractions were observed in healthy blood vessels, suggesting that they play a physiological role in the endothelium-dependent regulation of vascular tone.
However, endothelium-dependent contractions are also frequently associated with cardiovascular disease in both animals and by: 3. The hyperpolarizations caused by NO are usually prevented by blockers of ATP-dependent potassium-channels, which is not the case for endothelium-dependent hyperpolarizations caused by Cited by: Endothelium-dependent hyperpolarization induced by acetylcholine (ACh).
Typical tracings of the membrane potential (E m) recorded in smooth muscle cells from small gastric arteries during application of μM ACh in the presence (A, C) and the absence (B) of a functional endothelium (E) and after pre-exposure to 30 μM of Ba 2+, an Cited by: 3.
Book Description. Understanding the nature and role of endothelium-derived hyperpolarizing factor appears to be crucial in the quest for improved treatments for hypertension, diabetes, ischemia-reperfusion and other vascular disorders. EDHF comprises the proceedings of the Third International Symposium on endothelium-dependent hyperpolarizations.
EDHF contains the proceedings of the Fourth International Symposium on endothelium-dependent hyperpolarizations and will be of interest not only to physiologists and pharmacologists puzzled by the complexity of the interactions between the endothelium and underlying vascular smooth muscle cells, but also clinical researchers and physicians.
Endothelium-dependent hyperpolarizations Article in Drug Development Research 55(4) April with 7 Reads How we measure 'reads'.
EDHF comprises the proceedings of the Third International Symposium on endothelium-dependent hyperpolarizations. The first two meetings established a role for EDHF and other chemical factors involved in endothelium-dependent hyperpolarizations.
Like the release of nitric oxide, endothelium-dependent hyperpolarization apparently depends on calcium calmodulin, because inhibitors of this enzyme, calmidazolium and fendiline, decrease the endothelium-dependent hyperpolarization and relaxations of the porcine coronary artery to bradykinin, which are resistant to inhibitors of nitric oxide.
82 83Cited by: Researcher from all over the world joined this event and discussed signalling mechanisms in resistance vessels with emphasis on the importance of endothelium‐dependent hyperpolarizations. The meeting resulted in 11 publications in this issue Acta Physiologica Cited by: 1.
Endothelium-dependent relaxations can be mediated by the release of the endothelium-derived nitric oxide and prostacyclin . In addition, endothelial cells release a yet unidentified endothelium-derived hyperpolarizing factor (EDHF), which causes membrane hyperpolarization by opening potassium channels in vascular smooth muscle [ 3 - 4 ].Cited by: 1.
We examined endothelium-dependent and -independent hyperpolarizations and endothelium-dependent relaxation responses in carotid arteries isolated from streptozotocin-induced diabetic rats and age-matched controls.
The resting membrane potentials were not significantly different between control and diabetic carotid arteries. The endothelium-dependent hyperpolarization induced Cited by: The elucidation of the mechanism underlying endothelium-dependent hyperpolarizations and the discovery of specific inhibitors of the phenomenon are prerequisite for the understanding of the.
Acetylcholine induced endothelium‐dependent, transient hyperpolarizations and relaxations that were not affected by indomethacin.
Inhibition of the sodium‐potassium pump by ouabain or potassium‐free solution did not inhibit the relaxation to acetylcholine but prevented the corresponding hyperpolarization. by: Endothelium-Dependent Hyperpolarizations by Paul M. Vanhoutte (Editor) avg rating — 0 ratings — published.
Researcher from all over the world joined this event and discussed signaling mechanisms in resistance vessels with emphasis on the importance of endothelium-dependent hyperpolarizations. The meeting resulted in eleven publications in this issue Acta Physiologica which describe large findings in small blood by: 1.Under physiological conditions, the endothelium generates vasodilator signals [prostacyclin, nitric oxide NO and endothelium‐dependent hyperpolarization (EDH)], for the regulation of vascular tone.
The relative importance of these two signals depends on the diameter of the blood vessels: as the diameter of the arteries decreases, the Cited by: The altered endothelium‐dependent hyperpolarizations in diabetic arteries were almost completely prevented by insulin therapy.
Endothelium‐dependent relaxations by ACh in the presence of 10 −4 M L ‐NOARG and 10 −5 M indomethacin in diabetic arteries were Cited by: